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- publication . Preprint . Article . 2019Open Access EnglishAuthors:Alessia Landi; Muriel Mari; Tobias Wolf; Svenja Kleiser; Christine Gretzmeier; Isabel Wilhelm; Dimitra Kiritsi; Roland Thuenauer; Roger Geiger; Alexander Nyström; ...Persistent Identifiers
pmc: PMC6858607
pmid: 31732693
Publisher: Life Science Alliance LLCCountry: NetherlandsProject: EC | LEC&LIP2INVADE (282105), NWO | A three-dimensional look ... (2300175771), EC | DRIVE (765912), EC | PRONKJEWAIL (713660)Lectins are glycan-binding proteins with no catalytic activity and ubiquitously expressed in nature. Numerous bacteria use lectins to efficiently bind to epithelia, thus facilitating tissue colonisation. Wounded skin is one of the preferred niches for Pseudomonas aerugi...
Add to ORCID Please grant OpenAIRE to access and update your ORCID works.This research outcome is the result of merged research outcomes in OpenAIRE.
You have already added works in your ORCID record related to the merged research outcome. - other research product . Other ORP type . 2017Open Access EnglishAuthors:Cong, Yingying; Verlhac, Pauline; Reggiori, Fulvio;Project: NWO | A three-dimensional look ... (2300175771), EC | PRONKJEWAIL (713660), SNSF | ER-phagy mechanisms to ma... (CRSII3_154421)
Autophagy is a conserved intracellular catabolic pathway that allows cells to maintain homeostasis through the degradation of deleterious components via specialized double-membrane vesicles called autophagosomes. During the past decades, it has been revealed that numero...
- publication . Article . 2017Open Access EnglishAuthors:Yingying Cong; Pauline Verlhac; Fulvio Reggiori;Persistent IdentifiersCountry: NetherlandsProject: EC | PRONKJEWAIL (713660), NWO | A three-dimensional look ... (2300175771), SNSF | ER-phagy mechanisms to ma... (CRSII3_154421)
Autophagy is a conserved intracellular catabolic pathway that allows cells to maintain homeostasis through the degradation of deleterious components via specialized double-membrane vesicles called autophagosomes. During the past decades, it has been revealed that numero...
Add to ORCID Please grant OpenAIRE to access and update your ORCID works.This research outcome is the result of merged research outcomes in OpenAIRE.
You have already added works in your ORCID record related to the merged research outcome.